BRAIN AND NERVE INJURY WILL RECOVER,NO MORE PERMANENT LOSS

Prof Dr,DRAM,HIV /AIDS,HEPATITIS ,SEX DISEASES & WEAKNESS expert,New Delhi,India, +917838059592

Brain and Nerve Injury whether cause by trauma or by any metabolic or neuro degenerative disorders is always permanent as Neurons or cells of Nerves or  Brain never recovers.Similiary as we grow our grey matter or neurons are replaced by fibres or white matter or a network of Axons.That is the basis of so many degenerative disorders like dementia,memory loss ,Alzimer's disease and many mental retardations including Autism.

                        If Human being learn a process by which nerves particularly neurons are regenerated than Man will be never an old person,his memory and intellectuals all can be enhanced or supressed ,many neurological and psychiatric diseases will be easily treated.In this regard present research which has indentified an Enzyme which maintains nerve function particularly of Axon function is a pathway breaking study to treat so many yet to be resolved neuro degenerative diseases.                  

           Researchers have identified an enzyme that plays a crucial role in the degeneration of axons, the threadlike portions of a nerve cell that transmit signals within the nervous system.The research team discovered a new role of the enzyme Axundead -- or Axed -- in promoting self-destruction of axons.When Axed function was blocked, injured axons not only maintained their integrity but remained capable of transmitting signals within the brain`s complex circuitry for weeks, according to the findings published in the journal Neuron.
           "If you target this pathway, you have a really good chance of preserving the functional aspects of neurons after a variety of types of trauma or injury," said senior author Marc Freeman, Director of the Vollum Institute at Oregon Health & Science University in the US.It`s a very attractive therapeutic target," said Freeman, who conducted the work during his stint at the University of Massachusetts Medical School.

             "If we can find ways to block it, maybe we can preserve function in a wide array of patients who have lost axons through neurodegenerative diseases or other neural trauma," Freeman said.