Age Related Macular Degeneration Treatment In Ghatkopar, Mumbai
Posted by on Tuesday, 14th June 2022
Age Related Macular Degeneration
Human eye has various important parts like Cornea, Pupil, Iris, Lens and Retina. The macula is located in the center of the retina, the light-sensitive tissue at the back of the eye. The retina instantly converts light, or an image, into electrical impulses. The retina then sends these impulses, or nerve signals, to the brain. When the cells of the macula deteriorate, images are not received correctly. In early stages, macular degeneration does not affect vision. Later, if the disease progresses, people experience wavy or blurred vision, and, if the condition continues to worsen, central vision may be completely lost. People with very advanced macular degeneration are considered legally blind. Macular Degeneration is the leading cause of vision loss, more than cataracts and glaucoma combined.
Macular degeneration is classified as:
Dry Age related Macular Degeneration
Wet Age related Macular Degeneration.
Pathophysiology
The dry form is more common than the wet form, with about 85 to 90 percent of AMD patients diagnosed with dry AMD. The less common wet AMD usually leads to more serious vision loss.
Dry AMD causes changes of the retinal pigment epithelium, typically visible as dark pinpoint areas. The retinal pigment epithelium plays a critical role in keeping the cones and rods healthy and functioning well. Accumulation of waste products from the rods and cones can result in drusen, which appear as yellow spots. Areas of chorioretinal atrophy (referred to as geographic atrophy) occur in more advanced cases of dry AMD. There is no elevated macular scar (disciform scar), edema, hemorrhage, or exudation.
Dry AMD has three stages, all of which may occur in one or both eyes:
Early AMD - People with early AMD have either several small drusen or a few medium-sized drusen. At this stage, there are no symptoms and no vision loss.
Intermediate AMD - People with intermediate AMD have either many medium-sized drusen or one or more large drusen. Some people see a blurred spot in the center of their vision. More light may be needed for reading and other tasks.
Advanced AMD - In addition to drusen, people with advanced dry AMD have a breakdown of light-sensitive cells and supporting tissue in the central retinal area. This breakdown can cause a blurred spot in the center of your vision. Over time, the blurred spot may get bigger and darker, taking more of your central vision. You may have difficulty reading or recognizing faces until they are very close to you.
Wet AMD occurs when new abnormal blood vessels develop under the retina in a process called choroidal neovascularization (abnormal new vessel formation). Localized macular edema or hemorrhage may elevate an area of the macula or cause a localized retinal pigment epithelial detachment. Eventually, untreated neovascularization causes a disciform scar under the macula.
Symptoms
Dry macular degeneration symptoms usually develop gradually and without pain. They may include:
Visual distortions, such as straight lines seeming bent
Reduced central vision in one or both eyes
The need for brighter light when reading or doing close work
Increased difficulty adapting to low light levels, such as when entering a dimly lit restaurant
Increased blurriness of printed words
Decreased intensity or brightness of colors
Difficulty recognizing faces
What causes macular degeneration?
Though macular degeneration is associated with aging, there is genetic component to the disease. A strong association between development of AMD and presence of a variant of a gene known as complement factor H (CFH) is observed. This gene deficiency is associated with almost half of all potentially blinding cases of macular degeneration.
Other investigators have found that variants of another gene, complement factor B, may be involved in development of AMD.
Specific variants of one or both of these genes, which play a role in the body's immune responses, have been found in 74 percent of AMD patients who were studied. Other complement factors also may be associated with an increased risk of macular degeneration.
Oxygen-deprived cells in the retina produce a type of protein called vascular endothelial growth factor (VEGF), which triggers the growth of new blood vessels in the retina.
The normal function of VEGF is to create new blood vessels during embryonic development, after an injury or to bypass blocked blood vessels. But too much VEGF in the eye causes the development of unwanted blood vessels in the retina that easily break open and bleed, damaging the macula and surrounding retina.
Risk Factors
The biggest risk factor for Macular Degeneration is age. Your risk increases as you age, and the disease is most likely to occur in those 55 and older.
Other risk factors include:
Genetics – People with a family history of AMD are at a higher risk.
Race – Caucasians are more likely to develop the disease than African-Americans or Hispanics/Latinos.
Smoking – Smoking doubles the risk of AMD.
Diagnosis
AMD is detected during a comprehensive eye exam that includes:
Visual acuity test - This eye chart test measures how well you see at various distances.
Dilated eye exam - Drops are placed in your eyes to widen the pupils. Your eye care professional uses a special magnifying lens to examine your retina and optic nerve for signs of AMD and other eye problems. After the exam, your close-up vision may remain blurred for several hours.
Tonometry - An instrument measures the pressure inside the eye. Numbing drops may be applied to your eye for this test.
Both forms of age - related macular degeneration (AMD) are diagnosed by funduscopic examination. Visual changes can often be detected with an Amsler grid.
Color photography and fluorescein angiography are done when findings suggest wet AMD. Angiography shows and characterizes subretinal choroidal neovascular membranes and can delineate areas of geographic atrophy. Optical coherence tomography (OCT) aids in identifying intraretinal and subretinal fluid and can help assess response to treatment.
What Treatments Are Available for Macular Degeneration?
There’s no cure for macular degeneration. Treatment may slow it down or keep you from losing too much of your vision. Your options might include:
Lifestyle changes - like dieting, exercise, avoiding smoking, and protecting your eyes from ultraviolet light.
Anti-angiogenesis drugs - These medications – aflibercept (Eylea), bevacizumab (Avastin), pegaptanib (Macugen), and ranibizumab (Lucentis) -- block the creation of blood vessels and leaking from the vessels in your eye that cause wet macular degeneration. Many people who’ve taken these drugs got back vision that was lost. You might need to have this treatment multiple times.
Laser therapy - High-energy laser light can destroy abnormal blood vessels growing in your eye.
Photodynamic laser therapy - Your doctor injects a light-sensitive drug verteporfin (Visudyne) into your bloodstream, and it’s absorbed by the abnormal blood vessels. Your doctor then shines a laser into your eye to trigger the medication to damage those blood vessels.
Low vision aids - These are the devices that have special lenses or electronic systems to create larger images of nearby things. They help people who have vision loss from macular degeneration make the most of their remaining vision.
Submacular surgery - This removes abnormal blood vessels or blood.
Retinal translocation - A procedure to destroy abnormal blood vessels under the center of your macula, where your doctor can’t use a laser beam safely. In this procedure, your doctor rotates the center of your macula away from the abnormal blood vessels to a healthy area of your retina. This keeps you from having scar tissue and more damage to your retina. Then, your doctor uses a laser to treat the abnormal blood vessels.
Important Reminder: This information is only intended to provide guidance, not a definitive medical advice. Please consult eye doctor about your specific condition. Only a trained, experienced board certified eye doctor can determine an accurate diagnosis and proper treatment.
To schedule an appointment with our experts for Age Related Macular Degeneration Management please call us at +91 8451045935, +91-8451045934 or visit our clinic at Address.
For more information = https://www.mumbaieyecare.com/
Rate It
Posterior Vitreous Detachment Treatment In Ghatkopar, Mumbai
Posted by on Tuesday, 14th June 2022
Vitreous Detachment
The eye is a very complex functional and anatomic organ. The retina is a thin, delicate and transparent sheet of tissue that lines the inside of the back of the eye. Directly in front of the retina is also a cavity that contains a gel called vitreous. The structure responsible for the bulk and shape of our eye is Vitreous part. It is a jelly-like body that fills the posterior chamber of the eye, giving the eyeball its round shape and keeping the retina in place against the back of the eye.
It is made up of millions of tiny collagen fibrils along with ground substance mucopolysaccharides such as hyaluronic acid, which form a gel. The vitreous is mostly water, which makes up 98% of it. The collagen strands connect to the superficial layers of the retina especially around the macula, the retinal vessels or sites at the retinal periphery.
Posterior vitreous detachment (PVD), also known as hyaloid detachment, occurs when the retinal layer and vitreous body/posterior hyaloid membrane dissociate, with an intervening fluid collection forming in the subhyaloid space. It is thought to be a common consequence of aging, occurring in more than 70% of the population over the age of 60
Who is at risk of posterior vitreous detachment?
The risk factors for vitreous detachment include:
Older age.
Nearsightedness.
Past eye trauma.
Prior Cataract Surgery.
Vitreous detachment in one eye.
People over age 60 are more likely to develop vitreous detachment. But if you’re nearsighted or have suffered eye trauma, you’re more likely to develop it at a younger age. And if you’ve had vitreous detachment in one eye, you’re likely to experience it in the other eye within a year. It’s good to be aware when you’re at increased risk, then you’ll know to see an ophthalmologist promptly if new floaters and flashes develop.
Meanwhile, remember to safeguard your eyes. Wear protective goggles when you play sports, when you work with saws or other tools that create debris, and when you handle fireworks.
What are symptoms of Vitreous Detachment?
Posterior vitreous detachment (PVD) doesn’t cause pain or permanent vision loss, but you might experience other symptoms. They include:
Flashes. These small flashes of light are comparable to “seeing stars” after hitting your head. They can last a few seconds or minutes and tend to stop, or occur less often, once detachment is complete.
Floaters. These floating spots in your field of vision can resemble tiny specks, dust, dots, or cobweb-like shadows. They typically occur in the first few weeks of Posterior vitreous detachment (PVD) and are most noticeable when looking at a light surface, such as a white wall or the sky.
Cobweb effect. You may begin to see the outer edge of the vitreous as it separates from the retina. It can feel like you’re looking through a cobweb. This is temporary and goes away once detachment is complete.
How Vitreous Detachment Develops?
In normal eyes, the vitreous is attached to the surface of the retina through millions of tiny, intertwined fibers. Your vitreous gel is mostly made of water. As we age, the vitreous slowly shrinks, and these fibers pull on the retina's surface. If the fibers break, the vitreous can shrink further and separate from the retina, causing a vitreous detachment. Most people get Posterior vitreous detachment (PVD) at age 60 or older and it's very common after 80. It happens to men and women equally.
What other problems can vitreous detachment cause?
Vitreous detachment can sometimes lead to more serious eye conditions:
Retinal tear. Sometimes, the vitreous fibers tear a hole in the retina when they pull away. If you don’t get treatment quickly, this can lead to retinal detachment.
Retinal detachment. Sometimes vitreous detachment pulls the entire retina away from its normal position at the back of the eye. This can be a medical emergency. Learn more about retinal detachment.
Macular hole. Sometimes vitreous detachment tears a hole in the macula (the part of the retina that controls your central vision). This can happen before or after the vitreous detaches enough to cause floaters or flashes of light. Learn more about macular hole.
Macular pucker. Sometimes vitreous detachment causes a thin layer of scar tissue to grow over the macula. This usually happens slowly in the months or years after vitreous detachment. Learn more about macular pucker.
These conditions can cause vision loss but treatment may help preserve your vision. Tell your eye doctor right away if you notice symptoms of vitreous detachment so they can check for these more serious problems.
Diagnosis
A Routine eye examination can help your doctor to spot problems like Posterior vitreous detachment (PVD) early and that can help protect your vision.
Your doctor may use special eye drops to make your pupils (the holes in the center of your eyes) bigger and use a slit-lamp test to look for signs of Posterior vitreous detachment (PVD). This is done with a microscope that looks through the front of your eye. It is helpful in detecting, if Posterior vitreous detachment (PVD) has caused bleeding, a torn retina, or something else that could harm your eyesight.
Your doctor also may use other tests to make sure the gel hasn't pulled away from your retina. These include:
Optical coherence tomography (OCT)- a 3-D scan of the inside of your eye
Ocular ultrasound - a test that uses sound waves to show the inside of your eye
Treatment
Posterior Vitreous Detachment usually doesn’t require treatment.
Complete detachment typically takes no longer than three months. If you continue to see floaters after detachment is complete, discuss treatment options with your doctor.
When complications occur and your ophthalmologist recommends treatment, there are a number of options available, including:
Laser or cryosurgery: This can be done in the office with no anesthesia. Your doctor repairs the holes or tears in your retina, which prevents further progression of the condition.
Scleral buckle: This involves placing a band around the outside of the eye to counterbalance the force of the vitreous pulling on the retina. Fluid is then drained from behind your retina, allowing it to return to its proper position. This is usually done as outpatient surgery.
Pneumatic retinopexy: This surgery is sometimes done in the office. Your doctor injects a gas bubble into the vitreous behind your eye. The bubble pushes the tear against the back wall of the eye and closes it. Then your doctor uses laser or cryosurgery to secure the retina where it belongs. After surgery, you may need to keep your head in a certain position for a few days. The gas bubble dissipates over time.
Follow up of Vitreous Detachment
Once it has been confirmed that the vitreous detachment is isolated, follow-up examinations are recommended at regular intervals thereafter. The period between examinations depends, of course, on the presence of blood in the vitreous or other signs which could increase the likelihood of retinal detachment. Thus the first re-visit may be after a week or a month, according to the nature of the detachment.
Important Reminder: This information is only intended to provide guidance, not a definitive medical advice. Please consult eye doctor about your specific condition. Only a trained, experienced board certified eye doctor can determine an accurate diagnosis and proper treatment.
To schedule an appointment with our experts for Posterior vitreous detachment (PVD) Treatment In Ghatkopar, please call us at +91 8451045935, +91-8451045934 or visit our clinic at Address.
For more information = https://www.mumbaieyecare.com/
Rate It
Retinal Detachment Treatment From Retina Specialist In Mumbai - Dr. Jatin Ashar
Posted by on Tuesday, 7th June 2022
Retinal Detachment
Retina Detachment Treatment In Ghatkopar
Eye is made up of Iris, Pupil, Cornea and Retina. The retina is an extremely thin tissue that lines the inside of the back of the eye. It is the light-sensitive portion of the eye. Light from the objects we are looking at, enters the eye. Cornea and the eye lens focus the light image onto the retina. Human eye works like a camera, light striking the retina causes a complex biochemical change within certain layers of the retina and this, in turn, stimulates an electrical response within other layers of the retina.
These electric signals are transmitted by the nerve endings to the brain through optic nerve, which connects the eye to the brain. Within specific areas of the brain, this electrical energy is received and processed to allow us both to see and to understand what we are seeing. The retina has been compared to the film of a camera. However, once used, film has a permanent image on it. The neurosensory retina, in contrast, continually renews itself chemically and electrically, allowing us to see millions of different images every day without them being superimposed.
The retina is about the size of a postage stamp. It consists of a central area called the macula and a much larger peripheral area of the retina. The light receptor cells within the retina are of two types called the cones and the rods. Cones are concentrated within the macular (central) area and provide us with the sharpness of central vision and color vision. Rods predominate in the peripheral area of the retina and allow us to see in conditions of reduced illumination. The peripheral retina allows us to see objects on either side (peripheral vision) and, therefore, provides the vision needed for a person to move about safely.
Retinal Detachment
Retinal detachment occurs when the retina becomes separated from the nerve tissues and blood supply underneath it. While painless, visually this has a clouding effect that has been likened to a gray curtain moving across the field of vision.
There are 3 types of detachment: rhegmatogenous (which involves a retinal break), traction, and serous (exudative) detachment. Traction and serous retinal detachments do not involve a break and are called nonrhegmatogenous.
Rhegmatogenous detachment is the most common type and caused by a tear or hole in the retina. Risk factors include the following:
Myopia
Previous cataract surgery
Ocular trauma
Lattice retinal degeneration
A family history of retinal detachment
Traction retinal detachment can be caused by vitreoretinal traction due to preretinal fibrous membranes as may occur in proliferative diabetic or sickle cell retinopathy.
Serous detachment results from transudation of fluid into the subretinal space. Causes include severe uveitis, especially in Vogt-Koyanagi-Harada disease, choroidal hemangiomas, and primary or metastatic choroidal cancers (see Cancers Affecting the Retina).
Symptoms
A person with a detached retina may experience a number of symptoms.
These include:
Photopsia, or sudden, brief flashes of light outside the central part of their vision, or peripheral vision. The flashes are more likely to occur when the eye moves.
A significant increase in the number of floaters, the bits of debris in the eye that make us see things floating in front of us, usually like little strings of transparent bubbles or rods that follow our field of vision as our eyes turn. They may see what looks like a ring of hairs or floaters on the peripheral side of the vision.
A heavy feeling in the eye
A shadow that starts to appear in the peripheral vision and gradually spreads towards the center of the field of vision
A sensation that a transparent curtain is coming down over the field of vision
Straight lines start to appear curved
Diagnosis
Your doctor may use the following tests, instruments and procedures to diagnose retinal detachment:
Retinal examination. The doctor may use an instrument with bright light and special lenses to examine the back of your eye, including the retina. This type of device provides a highly detailed view of your whole eye, allowing the doctor to see any retinal holes, tears or detachments.
Ultrasound imaging. Your doctor may use this test if bleeding has occurred in the eye, making it difficult to see your retina.
Treatment
The goal of treatment is to re-attach the retina to the back wall of the eye and seal the tears or holes that caused the retinal detachment. Several approaches can be employed to repair a retinal detachment:
Scleral buckle - In this surgery, a silicone band is placed outside the eye wall to push the wall of the eye closer to the retinal tear in order to close the tear. The tear is treated with a freezing treatment to induce controlled scarring around the tear and permanently seal it. The fluid under the retina is sometimes removed at the time of surgery.
Vitrectomy - In this surgery, three small incisions are made in the white part of the eye and fine instruments are manipulated using an operating microscope to remove the vitreous gel that fills the eye and drain the fluid from under the retina. The surgeon may then use a laser or cryopexy to seal the retinal tears or holes. The eye is then filled with a gas bubble to hold the retina in place while it heals.
Pneumatic retinopexy - In this office-based procedure, a gas bubble is injected into the eye and the patient maintains a specific head posture to position the gas bubble over the retinal tear. The tear itself is sealed either with a freezing treatment at the time of the procedure, or with laser after the retina is re-attached.
Laser surgery - In certain cases, a retinal detachment can be walled off with laser to prevent the retinal detachment from spreading. This is generally appropriate for small detachments.
Complications after the surgery
Like any other surgery, retinal detachment procedures can also be followed by complications like:
Allergies to medications
Bleeding in the eye
Double vision
Cataracts
Glucoma
Eye infection
Chance that the retina does not reattach properly
Chance that the retina detaches again
Things to expect after surgery:
You might have some discomfort for a few a days to weeks after surgery. You will be given pain medicine to help you feel better.
You need to rest and be less active after surgery for a few weeks. Your ophthalmologist will tell you when you can exercise, drive or do other things again.
You will need to wear an eye patch after surgery. Be sure to wear it as long as your doctor tells you to.
If a bubble was put in your eye, you will need to keep your head in one position for a certain length of time, such as 1–2 weeks. Your doctor will tell you what that specific head position is. It is very important to follow the directions so your eye heals.
You might see floaters and flashing lights for a few weeks after surgery. You may also notice the bubble in your eye.
Your sight should begin to improve about four to six weeks after surgery. It could take months after surgery for your vision to stop changing. Also, your retina may still be healing for a year or more after surgery. How much your vision improves depends on the damage the detachment caused to the cells of the retina.
Important Reminder: This information is only intended to provide guidance, not a definitive medical advice. Please consult eye doctor about your specific condition. Only a trained, experienced board certified eye doctor can determine an accurate diagnosis and proper treatment.
To schedule an appointment with our experts for Retinal Detachment Treatment In Ghatkopar, please call us at +91 8451045935, +91-8451045934 or visit our clinic at Address.
For more information = https://www.mumbaieyecare.com/
Rate It
Corneal Arcus or Arcus senilis : Causes, Symptoms, Diagnosis, and Treatment at mumbaieyecare
Posted by on Tuesday, 7th June 2022
Corneal Arcus
Corneal Arcus or Arcus senilis appears as a white, gray, or blue ring or arc around the cornea of the eye. The condition is usually seen in older adults but can affect people of all ages, even appearing at birth. Arcus senilis is generally harmless, and an involutional change modified by genetic factors. However, arcus is sometimes indicative of hyperlipidoproteinemia (involving low-density lipoproteins) with elevated serum cholesterol, especially in patients under 40 years of the age.
Causes of corneal arcus
Arcus is a deposition of lipid in the peripheral corneal stroma. Cholesterol and triglycerides are two types of fats in your blood. Some of the lipids in your blood come from foods you eat, such as meat and dairy products. Your liver produces the rest. Lipid deposition starts at the inferior and superior poles of the cornea and in the late stages encircle the entire circumstances. Arcus senilis is more common in men than in women. In people under age 40, arcus senilis is often due to an inherited condition and in rare cases, children are born with arcus senilis ( arcus juvenilis).
Arcus senilis can also appear in people with Schnyder central crystalline dystrophy. This rare, inherited condition causes cholesterol crystals to deposit on the cornea.
Symptoms
A person with arcus senilis may develop:
A white, gray, or blue circle around the cornea of the eye.
The circle or arc will have a sharp outer border but a blurred inner border.
If someone has an arc, the lines could grow to form a complete circle in front of the iris.
The presence of corneal arcus in patients younger than 40 year-old warrants evaluation for systemic lipid abnormalities.
Unilateral arcus is a rare condition associated with contra lateral artery disease or ocular hypotony. Usually it is an asymptomatic condition and does not affect your vision.
Diagnosis
Eye examination performed by eye doctor can diagnose arcus senilis. A slit lamp microscope is a very helpful tool to evaluate eye thoroughly. A special eye drop is used to widen the pupil of the patient. Once the eye drops works, ophthalmologist can inspect all parts of the eye with back of the eye as well for any disease. In arcus, thickness of the vessels increases due to fat deposition. Surgeon has to look for signs of atherosclerosis also, which is a condition where arteries become clogged with fatty substances.
A blood test will determine whether someone has high cholesterol. If they do, they may be prescribed medicine or advised on a suitable diet and exercise program to lower the cholesterol in their blood.
How corneal arcus is managed?
The good news is, you don’t need to treat arcus senilis. It typically affects both eyes, but in some cases only one eye has a grayish arc around the cornea, which may be a sign of poor blood circulation.
Eating healthy foods and exercising regularly are some good ways to keep your eyes healthy. We recommend wearing sunglasses with 100% UV protection when outdoors and safety goggles when doing hazardous tasks as well.
If arcus senilis is a sign of high cholesterol, a doctor may recommend a diet that is low in saturated fats and high in fruit, vegetables, and fiber.
If lipid levels are not controlled by diet and exercise are not enough, several medications can help lower your lipid levels:
Statin drugs block a substance your liver uses to make cholesterol. These drugs include atorvastatin, fluvastatin, pravastatin and rosuvastatin.
Bile acid binding resins force your liver to use more cholesterol to produce digestive substances called bile acids. This leaves less cholesterol in your blood. These drugs include cholestyramine, colesevelam and colestipol.
Cholesterol absorption inhibitors like ezetimibe reduce your body’s absorption of cholesterol.
Drugs may be used to lower triglyceride levels:
Fibrates reduce production of lipids in your liver and increase the removal of triglycerides from your blood.
Niacin reduces the production of lipids by your liver.
Increased exercise and quitting smoking can also help.
Possible complications
Arcus senilis alone is not known to have any complications unless it is a sign of high cholesterol. High levels of cholesterol in a person’s blood can cause significant problems, such as coronary artery disease or cardiovascular disease.
Follow-Up
Arcus senilis may sometimes appear as a white ring around the iris due to calcium deposits on the circumference of the cornea, where the white section of your eye (the sclera) and the colored part (the iris) meet (called the limbus). When the deposits consist of calcium, the limbus appears milky and may indicate tissue damage that needs correction.
The blue (or sometimes gray) ring around the iris often is associated with high cholesterol and triglycerides. In turn, this may be an indicator of heart condition that yor are more prone to heart attacks and strokes. When this condition appears in people younger than 60 years old, regular blood tests are normally recommended to monitor elevated lipid levels.
In few other cases, when corneas become opaque, due to genetic mutation it could be from a lowered high-density lipoprotein (HDL) cholesterol level. This condition can result in other eye problems. Principally, this opacification may be related to deficient levels of lecithin cholesterol acyl transferase (LCAT). LCAT is an enzymatic protein responsible for converting free cholesterol into cholesteryl ester.
Cholesterol ester, is then separated into the nucleus of the lipoproteins to produce a synthetic high-density lipoprotein (HDL) chain. This reaction is forced one way, since the deposits are removed from the exterior. So the enzyme is attached to both HDLs and low-density lipoproteins (LDLs) in blood plasma.
This process can result in the manifestation of fish eyes, and it is actually called “fish eye disease.” Corneal opacification can also result from mutations in another HDL protein, the adenosine triphosphate-binding protein ABCA1.
Just because you are younger than 40 and have arcus does not mean you absolutely have elevated lipid levels, but at a very minimum, it is recommended that you have them checked.
Unilateral corneal arcus may be associated with vascular or inflammatory etiologies.
Important Reminder: This information is only intended to provide guidance, not a definitive medical advice. Please consult eye doctor about your specific condition. Only a trained, experienced board certified eye doctor can determine an accurate diagnosis and proper treatment.
To schedule an appointment with our experts for Corneal Arcus Treatment In Ghatkopar, please call us at +91 8451045935, +91-8451045934 or visit our clinic at Address.
For more information = https://www.mumbaieyecare.com/
Rate It
Dry Eye Treatment In Ghatkopar, Eye Specialist in Ghatkopar - Mumbai Eye Care
Posted by on Tuesday, 31st May 2022
Dry Eyes Treatment In Ghatkopar
A dry eye is an uncomfortable and painful condition. It can be understood as an inadequate production of tears leading to dryness of eyes. Dry eyes make you more susceptible to bacterial infections and trauma due to reduced lubrication over the cornea. As tear film is very thin, reading, writing and watching becomes very uncomfortable.
Role of tears
Blinking makes your eyes moist every time and keeps the corneal surface smooth and clean. Tear film contains a combination of oil, water and mucous. Outermost layer is made up of oil as it prevents tear from drying. Second layer (water) comes from lacrimal gland and made up of mostly water as main content. Water washes away any dirt and unwanted particles from the eye to keep it clean. Inner most layer is made up of mucus and formed in conjunctiva, due to this layer tears don’t stick to eyes. In general we see with any irritant, our eyes protective response is to produce tears but in few local and system medical conditions, tear formation is reduced and that hampers with the vision.
Why do I get dry eye condition?
We produce tears all the time, not only when we are emotional but with every blink. Dry eye can be because of various reasons:
Altered composition of tears or tears dry up too fast – imbalance is oil, water and mucous content may result in dry eyes.
Inflammation or trauma to the eye (lacrimal gland, conjunctiva) – dry eyes may result as a consequence to damaged tear producing apparatus.
Medical conditions for less production of tears – autoimmune diseases like lupus, scleroderma, rheumatoid arthritis, Parkinsons disease.
Vitamin A deficiency may result in poor eye health
Disease like diabetes or radiation exposure also affects eye status.
Hormonal imbalance like pregnancy or menopause also reduces tear production.
Drugs like Antihistamins, antidepressants may result in dry eyes.
Corneal nerve desensitivity caused by contact lens or nerve damage by laser eye surgery.
As an aging process.
Other medical conditions can lead to dry eyes, such as the following:
Keratoconjuctivitis - It refers to the inflammation of the surface of your eye, called the cornea or the conjunctiva
Keratitis is another condition that’s caused by irritation or swelling of your cornea when your eyes are dry for too long
Keratoconjuctivitis Sicca – This term is used to describe an autoimmune condition when you aren’t able to produce enough tears and develop an infection or inflammation.
How do I know that I have dry eyes condition?
Symptoms experienced by patients suffering from dry eyes are:
Fatigued and heavy eyes
Sore and itchy eyes
Burning and dryness sensation
Red eyes with blurred vision
Difficulty in wearing contact lenses
Difficulty in night driving
Sensitivity to light and stringy mucous around the eyes
Eyelids sticking together when waking up after sleep
Double vision
How diagnosis is made?
Ophthalmologist conducts a thorough eye examination and certain tests to reach to a final conclusion and design the treatment plan.
Detailed Medical and drug history of systemic diseases may help in diagnosis.
History of trauma and family occurrence can be related to present condition.
Schirmer test – this test is performed to see the volume of your tears, blotting strips of paper are placed under your lower eyelids. After five minutes your doctor measures the amount of strip soaked by your tears.
Phenol red thread test - In this test, a thread filled with pH-sensitive dye (tears change the dye color) is placed over the lower eyelid, wetted with tears for 15 seconds and then measured for tear volume.
Special dyes are used for your eye doctor to check the evaporation of your tears. As tear composition has oil content in it, so if it is reduced, it may lead to quick drying of your tears.
Tear osmolarity test can be done to measure the composition of particles and water in your tears.
Special Tear markers for dry eye disease like matrix metalloproteinase-9 or lactoferrin.
What are my treatment options?
Treatment is purely based on the cause as it may range from behavior modifications to medical procedures.
Behavior modifications like adjustment of your computer screens below eye level, taking short breaks between long tasks. Avoid harsh environments like dry winds, driving without protective eyeglasses; Sunglasses are must on sunny days.
Artificial tears – ophthalmologists prescribe you artificial tears, their composition is just like natural tears. If you are using more than six times a day, preservative free tears are also available in the market.
Treating local factors - warm compresses to the eyes, eyelid cleaners and massaging your eyelids.
Medication to reduce inflammation and infection to the eyes
Tear-stimulating drugs. Drugs called cholinergics (pilocarpine, cevimeline) help increase tear production. These drugs are available as pills, gel or eyedrops
Eye inserts that work like artificial tears. If you have moderate to severe dry eye symptoms and artificial tears don't help, another option may be a tiny eye insert that looks like a clear grain of rice.
Unblocking oil glands - improves the composition of the tears.
Punctal plugs – removal punctal plugs are tiny silicon plugs used to block the tear ducts to prevent tear loss. Punctal occlusion can be done by cautery.
Surgery – sometimes problems like incomplete blinking is treated with surgical procedure by oculoplastic surgeon (specialist in eyelid problems).
Lipiflow - This medical device uses heat and pressure to unclog blocked glands on your eyelids.
Salivary gland transplantation – It is a surgical procedure that is occasionally considered in persistent and severe cases that have not responded to other treatments.
Light therapy - Using intense pulsed light therapy followed by massage.
Prevention
As an aging process, tears production will eventually go down, but still precautions may help to avoid dry eyes discomfort.
Add humidifier to your room in winters
At high altitudes or extreme winters, take care of your eyes as dry winds may be traumatic to your eyes and tear producing apparatus.
If you are a person spending more time on computer or phone, take short brakes during your tasks as your eyes get tired by long focus and radiation exposure.
Use sunglasses while going outdoors
Use of ointments instead of drops
Frequent washing of your eyes also serves a natural way for eye moisture and cleaning.
Mild soap use to reduce the irritation.
Important Reminder: This information is only intended to provide guidance, not a definitive medical advice. Please consult eye doctor about your specific condition. Only a trained, experienced board certified eye doctor can determine an accurate diagnosis and proper treatment.
To schedule an appointment with our experts for Dry Eye Treatment in Ghatkopar, please call us at +91 8451045935, +91-8451045934 or visit our clinic at Address.
For more information = https://www.mumbaieyecare.com
Rate It
Why we are No. 1 › 
Category (
Views ( 1067 ) | User Rating
